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《不同油菜品種對菌核病抗病性差異的生理生化分析》由會員上傳分享，免費在線閱讀，更多相關(guān)內(nèi)容在學(xué)術(shù)論文-天天文庫。
1、thanweakpathogenicstrainWW2．Thisresultwasconsistentwiththeseedlingstage．TheactivityofCATwasshowingatrendthatdecreasedfirstandthenincreasedbothinresistantandsusceptiblevarietiesafterinoculation，resistantvarietieswerehigherthatofsusceptiblevarietiesandconsistentlylowert
2、hatofcontrol，resistantvarietiesdecreasedgreatlyandsusceptiblevarietiesdecreasedwithsmallchangeranges．StrongpathogenicstrainNG4inducedthereductionofCATactivityinalargerchangerangeweakpathogenicstrainWW2andreachedsignificantdifference．PALactivityoftheresistantandsuscept
3、iblevarietieswerebothincreasedandthendecreasedafterinoculatedwithstrongpathogenicstrainNG4，resistantvarietiesproducedtwowavecrests，susceptiblevarietieschangeslightly．PALactivityinresistantvarietiesincreasedearliersusceptiblevarieties，accumulatedmore．PALactivityofallfo
4、urvarietiesshowedatrendofincreasedandthendecreasedafterinducedwithweakpathogenicstrainWW2，buttherateofincreaseinresistantvarietieswasgreatersusceptiblevarieties．Itexplainedthatthereweredifferentresistancemechanismsofdifferentresistantvarietiesofoilseedrapetostrainsofd
5、ifferentpathogenicity．Chitinaseactivityofallvarietiesincreasedafterinoculation，resistantvarietiesrisefasterandlastforalongertimethesusceptibleones，resistantvarietiesreachedthewavecrestearlierthesusceptibleones，itexplainedthatSsclerotiorumwasabletoinducetheproductionof
6、chitinase．Therewasapositivecorrelationbetweenraperesistanceanditschitinaseactivity．ChitinaseactivityinducedbystrongpathogenicstrainNG4washigherweakpathogenicstrainWW2．InhibitionofchitinasetomycelialgrowthofS．sclerotiorum：chitinasecrudeenzymeextractedfrominfectedrapele
7、avescouldinhibitedmyceliumgrowthofS．sclerotioruminvitro．however,＆sclerotiorummycelialcontinuedtogrowthinthecontrolarea．ItshowedthatchitinasehadacertainantibacterialactivitytoSsclerotiorum．Keywords：Sclerotiniaselerotiorum；diseaseresistance；oxalicacid；biochemicalmechani
8、sm；IV目錄摘要????????????????????????????????．．IABSTRACT????????????????????????????????????????III文獻綜述?????????????????????????
